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Molecular Cell Biology
Neeraja Sankaran | Sep 17, 1995 | 2 min read
A. Noda, Y. Ning, S.F. Venable, O.M. Pereira-Smith, J.R. Smith, "Cloning of senescent cell-derived inhibitors of DNA synthesis using an expression screen," Experimental Cell Research, 211:90-8, 1994. (Cited in nearly 150 publications through August 1995) Comments by James R. Smith, Baylor College of Medicine The major finding described in this paper, says James R. Smith, a professor in the division of molecular virology at Baylor College of Medicine and codirector of the Roy M. and Phyllis Gou
Molecular Biology
The Scientist Staff | Aug 22, 1993 | 2 min read
M. Leid, P. Kastner, R. Lyons, et al., "Purification, cloning and RXR identity of the HeLa cell factor with which RAR or TR heterodimerizes to bind target sequences efficiently," Cell, 68:377-95, 1992. Mark Leid (Oregon State University, Corvallis): "The diverse effects of retinoic acid (RA) on development, cellular growth and differentiation, and homeostasis are mediated by two families of RA receptors that arose independently during evolution and belong to the steroid/thyroid hormone super
Molecular Biology
The Scientist Staff | Jan 6, 1991 | 1 min read
L. Osborn, C. Hession, R. Tizard, C. Vassallo, et al., "Direct expression cloning of vascular cell adhesion molecule 1, a cytokine-induced endothelial protein that binds to lymphocytes," Cell, 59, 1203-11, 22 December 1989. Laurelee Osborn (Biogen Inc., Cambridge, Mass.): "Vascular cell adhesion molecule 1 (VCAM1) is of interest to scientists in several disciplines, particularly those studying cell-cell adhesion mechanism and the mechanisms by which the inflammatory response begins and progre
Molecular Biology
The Scientist Staff | Jan 20, 1991 | 3 min read
C.K. Glass, S.M. Lipkin, O.V. Devary, M.G. Rosenfeld, "Positive and negative regulation of gene transcription by a retinoic acid-thyroid hormone receptor heterodimer," Cell, 59, 697-708, 17 November 1989. Christopher K. Glass (School of Medicine, University of California, San Diego, La Jolla): "Retinoic acid receptors appear to exert profound effects on vertebrate development by binding to target genes and altering the rates at which they are transcribed in response to retinoic acid. Because t
Molecular Biology
The Scientist Staff | Mar 3, 1991 | 1 min read
J.H. Exton, "Signaling through phosphatidylcholine breakdown," The Journal of Biological Chemistry, 265, 1-4, 5 January 1990. John H. Exton (Vanderbilt University School of Medicine, Nashville, Tenn.): "This article was designed to alert readers to the existence of what appears to be a novel cellular signaling system involving the breakdown of phosphatidylcholine to phosphatidic acid. It arose out of the realization that the accumulation of phosphatidic acid in stimulated cells could not be e
Molecular Biology
The Scientist Staff | Jan 19, 1992 | 1 min read
S. Ghosh, A.M. Gifford, L.R. Riviere, P. Tempst, et al., "Cloning of the p50 DNA binding subunit of NF-kB: homology to rel and dorsal," Cell, 62:1019-29, 1990. Sanker Ghosh (Yale University School of Medicine; formerly at Whitehead Institute for Biomedical Research, Cambridge, Mass.): "NF-kB is a transcription factor that is widely used for the inducible expression of a large number of cellular and viral genes. This has resulted in the study of NF-kB by researchers interested in diverse areas
Molecular Biology
The Scientist Staff | Jan 6, 1991 | 1 min read
B. Berkhout, R.H. Silverman, K.-T. Jeang, "Tat trans-activates the human immunodeficiency virus through a nascent RNA target," Cell, 59, 273-82, 20 October 1989. Ben Berkhout (National Institute of Allergy and Infectious Diseases, NIH, Bethesda, Md.): "The unique feature of this study was the use of RNA-refolding mutagenesis. Rearrangement of RNA structures was first described by Charles Yanofsky [Stanford University] to explain the process of transcriptional attenuation ["Transcription atten
Molecular Biology
The Scientist Staff | Dec 6, 1992 | 2 min read
R. Glynne, S. H. Powis, S. Beck, A. Kelly, L-A. Kerr, J. Trowsdale, "A proteasome-related gene between the two ABC transporter loci in the class II region of the human MHC," Nature, 353:357-360, 1991. Richard Glynne (Imperial Cancer Research Fund, London, England): "The mechanism by which the body's immune system recognizes and kills virally infected cells but passes over healthy cells has intrigued immunologists for many years. An important breakthrough came when Alain Townsend at the John R
Molecular Biology
The Scientist Staff | Jul 5, 1992 | 1 min read
S.J. Baker, S. Markowitz, E.R. Fearon, J.K.V. Willson, B. Vogelstein, "Suppression of human colorectal carcinoma cell growth by wild- type p53,"Science, 249:912-15, 1990. S.J. Baker, A.C. Preisinger, J.M. Jessup, et al., "p53 gene mutations occur in combination with 17p allelic deletions as late events in colorectal tumorigenesis," Cancer Research, 50:7717-22, 1990. Bert Vogelstein (Johns Hopkins Oncology Center, Baltimore, Md.): "Cancer represents not one disease but hundreds. Accordingly,
Molecular Biology
The Scientist Staff | Mar 1, 1992 | 2 min read
D.W. Nebert, D.R. Nelson, M.J. Coon, R.W. Estabrook, et al., "The P450 superfamily: update on new sequences, gene mapping, and recommended nomenclature," DNA and Cell Biology, 10:1-14, 1991. Daniel W. Nebert (University of Cincinnati Medical Center): "This review is the third in a series of comprehensive, up- to-date compilations of data about members of the large cytochrome P450 gene superfamily. It serves to organize a large--and growing--body of sequencing and mapping data on 154 P450 genes

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