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The danger of misfolded proteins

By | April 9, 2002

Protein folding intermediates are cytotoxic, independent of cell damage caused by mature folded proteins.

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Thick skinned

By | April 9, 2002

Lipoid proteinosis is caused by mutations in the extracellular matrix protein 1 gene on chromosome 1q21.

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BAC to BAC

By | April 8, 2002

Spotting modified DNA directly onto untreated glass surfaces offers an efficient system for identifying genomic abnormalities in tumor samples.

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Bop controls ventricle formation

By | April 8, 2002

encodes a muscle-restricted protein essential for cardiac differentiation and right ventricle morphogenesis.

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primase inhibitors may treat herpes

By | April 8, 2002

primase inhibitors are orally available drugs active against herpes simplex virus.

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Send in the clones

By | April 8, 2002

The US patent office has waded in to the dispute over who was first to develop cloning technology.

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Gene therapy rebuilds immunity and its image

By | April 5, 2002

Researchers at Great Ormond Street Children's Hospital strengthen the case for gene therapy as a treatment for immunodeficiencies.

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New vaccine hopes for cancer

By | April 5, 2002

Tumor cells covered with a T cell ligand-mimicking antibody fragment can initiate their own destruction.

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The genome that feeds the world

By | April 5, 2002

Two independent groups report draft sequences of the rice genome.

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The silencing team

By | April 5, 2002

Inactivation of tumor suppressor genes is an essential stage in the development of human cancer but the extent of the involvement of epigenetic silencing and hypermethylation in this process remains unclear. In April 4 Nature, Ina Rhee and colleagues from Johns Hopkins University School of Medicine, show that two enzymes (DNMT1 and DNMT3b) cooperate to silence genes and maintain DNA methylation in human cancer cells.Rhee et al. worked on a colorectal cancer cell line and observed that only genet

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