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Insights into paracetamol poisoning

By | November 8, 2000

Glutathione S-transferases (GST) may provide a means for treating fatal liver damage caused by paracetamol overdose. In a paper published in 7 November Proceedings of the National Academy of Sciences, Henderson et al provide a new insight into how the enzyme GST Pi may moderate the toxic effects of paracetamol (acetaminophen).When overdoses of paracetamol are taken, too much of the compound N-acetyl-p-benzoquinoneimine (NAPQI) is produced; it covalently binds to proteins and other macromolecules

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An association between phenylpropanolamine and stroke has prompted the US FDA to pull cold medicines from the market.

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Therapeutic cloning endorsed by Royal Society

By | November 8, 2000

The UK's Royal Society yesterday released a report backing continued research into the use of cloned embryonic stem cells.

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Architectural role for BCL6

By | November 7, 2000

Nuclear BTB/POZ proteins are often concentrated into discrete nuclear subdomains, but the role of these nuclear compartments is unclear. The BCL6 proto-oncogene, frequently altered in non-Hodgkin lymphoma, encodes a POZ/zinc finger protein that shows a characteristic localization in nuclear aggregates. In the November Molecular and Cellular Biology Albagli et al. used a tetracycline-regulated, epitope-tagged BCL6 allele to explore the significance of BCL6 aggregates (Mol Cell Biol 2000, 20:8560-

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Creating kingdoms

By | November 7, 2000

Analysis of four conserved proteins allows a better prediction of eukaryotic phylogeny.

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Life after ESTs

By | November 7, 2000

Expressed sequence tags (ESTs) have given researchers a quick if dirty look at the coding potential of the human genome. But now in the November Nature Genetics, Penn et al. use microarray experiments to conclude that the human genome project will uncover many genes not previously discovered by EST sequencing (Nat Genet 2000, 26:315-318). They scan 350 Mb of finished and draft human sequence using three different gene-finding algorithms. Open reading frames (ORFs) predicted by at least two of th

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HOUSTON. Amgen, the largest biotechnology company in the world, is funding research focusing on disorders that destroy parts of the nervous system. These include Alzheimer's disease, Parkinson's disease, multiple sclerosis and stroke. Medicines for disorders resulting from dysfunction of the neuroendocrine system such as obesity and Type 2 diabetes are also under development. In both areas, Amgen licenses product candidates and technologies that complement its internal drug discovery and develop

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New companies to commercialise neuroscience discoveries

By | November 3, 2000

Neurogenomics has significant commercial potential by way of gene targets for enhancement of brain function and treatment of brain-based disease.

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Fishy mutations

By | November 2, 2000

Transgenic animals carrying a prokaryotic vector are useful tools for mutation studies and the detection of spontaneous or induced mutations in different tissues. Advances in fish transgenesis make it possible to develop fish that can be used both to assess the health hazards of mutagens in aquatic environments and for comparative mutagenesis analysis. In a paper published online ahead of print in Proceedings of the National Academy of Sciences, Winn et al. report the use of a bacteriophage lamb

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Senior scientists promise to boycott journals

By | November 2, 2000

Leading scientists will refuse to publish, edit or subscribe to journals that do not make research articles available free of charge.

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