Making macrophages in atherosclerotic plaques digest spent organelles instead of dying may help keep plaques stable.
Making macrophages in atherosclerotic plaques digest spent organelles instead of dying may help keep plaques stable.
Using tiny lipid discs, scientists resolve contradictory evidence about how many proteins are required for neurotransmitter release.
Actin filaments respond to pressure by forming branches at their curviest spots, helping resist the push.
Two proteins interact to save adhesion molecules from degradation, potentially contributing to a more aggressive cancer.