Making macrophages in atherosclerotic plaques digest spent organelles instead of dying may help keep plaques stable.
Making macrophages in atherosclerotic plaques digest spent organelles instead of dying may help keep plaques stable.
Actin filaments respond to pressure by forming branches at their curviest spots, helping resist the push.
EDITOR'S CHOICE IN MICROBIOLOGY The paper S. Alsford et al., “High-throughput decoding of antitrypanosomal drug efficacy and resistance,” Nature, 482:232-36, 2012. The finding Trypanosoma brucei, the single-cell protozoan that causes the tropical
A cytokine involved in suppressing the immune system may actually activate it to kill cancer cells.
Two proteins interact to save adhesion molecules from degradation, potentially contributing to a more aggressive cancer.