Clinicians and researchers teamed up to investigate how inappropriate proinflammatory mechanisms contribute to the pathogenesis of drug-refractory epilepsy.

Clinicians and researchers teamed up to investigate how inappropriate proinflammatory mechanisms contribute to the pathogenesis of drug-refractory epilepsy.

Research in mice suggests that moderating nerve activity with drugs or electrical pulses could modify tissue immune responses, curtailing the chronic pain often associated with inflammatory conditions.

A gene essential for information storage in the brain engages an autoregulatory feedback loop to consolidate memory.

When children need medications, getting the dosing and method of administration right is like trying to hit a moving target with an untried weapon.

The body’s own mechanism for dispersing the inflammatory reaction might lead to new treatments for chronic pain.

Abnormal sympathetic neuron growth leads to simultaneous activation of clusters of sensory neurons, causing the difficult-to-treat sensation.

Researchers created a new high-throughput tool to hunt for therapies that remyelinate the nervous system.

The FDA has given the green light to the first major new classes of antidepressant therapies in decades, opening up new avenues for therapeutic development.