inflammation, immunology, neuroscience

Clinicians and researchers teamed up to investigate how inappropriate proinflammatory mechanisms contribute to the pathogenesis of drug-refractory epilepsy.

The temporary loss of olfaction stops the flies from eating any more of whatever it is that made them sick.

Knocking out the receptor for a lipid that causes inflammation rejuvenates macrophage metabolism and restores cognitive function in an Alzheimer’s disease model.

The once fringe idea is gaining traction among the scientific community.

Emerging evidence links bacterial or viral infection with the neuropathology of Alzheimer’s disease.

A mouse study suggests a mechanism by which severe infections during pregnancy increase autism risk. 

Not all inflammation leads to pain. Despite widespread infection followed by fever, colds rarely cause pain. But when some cytokines and certain immune cells are active near pain-sensing nerves, they trigger receptors that convey pain sensations to the brain.

The body’s own mechanism for dispersing the inflammatory reaction might lead to new treatments for chronic pain.

Nerve signals control T cell responses, helping to explain inflammation and stroke.