Researchers find that temporary double-stranded DNA breaks commonly result from normal neuron activation—but expression of an Alzheimer’s-linked protein increases the damage.
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As reproductive tissues age, DNA repair mechanisms become less efficient, causing genomic damage to accumulate.
Human cytomegalovirus fixes its broken DNA by exclusively co-opting its host’s repair proteins.
Researchers identify two new DNA repair systems, in addition to four that were already known, that can attack unprotected telomeres.
A handful of life science researchers will take home the United States' top science honor.
Is DNA damage an inevitable consequence of epigenetic reprogramming?
The minority of Parkinson’s cases now known to have genetic origins are shedding light on the cellular mechanisms of all the rest, bringing researchers closer to a cause—and perhaps a cure.
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