Researchers find that temporary double-stranded DNA breaks commonly result from normal neuron activation—but expression of an Alzheimer’s-linked protein increases the damage.
Researchers find that temporary double-stranded DNA breaks commonly result from normal neuron activation—but expression of an Alzheimer’s-linked protein increases the damage.
Protein aggregates in the brains of some people with dementia or motor neuron disease have a surprising origin.
Scientists use virus-free gene therapy on patient-derived stem cells to repair spinal muscular atrophy in mice.
An NIH study finds that the former NFL linebacker who committed suicide last May had signs of degenerative brain disease.
Misfolded α-synuclein proteins promote the spread of Parkinson’s pathology in mouse brains.
Older foraging bees experience a change in brain chemistry when they revert to nest duties typically given to younger individuals.
After 20 years of high-profile failure, gene therapy is finally well on its way to clinical approval.
A new study suggests that the brain injuries suffered by soldiers in Afghanistan may be similar to those observed in some athletes.
Delivering a missing enzyme to the brains of paralyzed children with a rare, life-threatening neurological disease restores movement and builds muscle mass.
Swedish researchers have discovered that patients with the neurodegenerative disorder had half the normal expected risk of developing tumors.