Making macrophages in atherosclerotic plaques digest spent organelles instead of dying may help keep plaques stable.
Making macrophages in atherosclerotic plaques digest spent organelles instead of dying may help keep plaques stable.
A roundup of recent research announced last weekend at the annual conference of the American Association for the Advancement of Science (AAAS).
There are five steps of autophagosome biogenesis: induction, expansion, vesicle completion, fusion, and cargo degradation. While much has been learned over the past few decades about how this process works, the big question that remains is where the
Despite years of research, the longstanding mystery of where the autophagosome gets its double lipid bilayers is not much clearer.
Autophagy, the cell’s recycling system, may be responsible for the health benefits of exercise.
Cellular structures known as midbodies, formed during cell division, appear to accumulate in stem cells and cancer cells, hinting at a potential function for these once-disregarded organelles.
Starving brain cells can stimulate hunger through a common cannibalistic act, possibly explaining why some dieters can’t resist temptation.
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