Researchers identify a protein involved in the chromosomal disorder that could explain its characteristic learning deficits.
Advertisement
The Scientist
Most Recent
Researchers find that temporary double-stranded DNA breaks commonly result from normal neuron activation—but expression of an Alzheimer’s-linked protein increases the damage.
Newly constructed ramps will expand the habitat available to a colony of water voles in London, and similar ramps elsewhere could encourage isolated populations to mix.
Improvements in light-sheet microscopy enable real-time activity imaging of almost every neuron in the brain of zebrafish larvae.
Native Australian frog tadpoles outcompete the tadpoles of the invasive cane toad, suggesting the native frogs could form part of a suburban control program.
Mice with human brain cells showed enhanced synaptic plasticity and learning, suggesting glia may be key to our cognitive prowess.
Normal proteins with regions resembling disease-causing prions are responsible for an inherited disorder that affects the brain, muscle, and bone.
Inducing certain brain patterns extends non-REM sleep in mice.
Children with dyslexia have an easier time learning to read after playing action video games that don’t incorporate reading.
Blind tadpoles regain vision when new eyes are grafted onto their tails.
Follow The Scientist
Stay Connected with The Scientist
-
The Scientist Magazine
-
The Scientist Careers
-
Neuroscience Research Techniques
-
Genetic Research Techniques
-
Cell Culture Techniques
-
Microbiology and Immunology
-
Cancer Research and Technology
Current Issue
