The Scientist

» artificial selection, ecology and immunology

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Contributors

By | April 1, 2016

Meet some of the people featured in the April 2016 issue of The Scientist.

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image: Guts and Glory

Guts and Glory

By | April 1, 2016

An open mind and collaborative spirit have taken Hans Clevers on a journey from medicine to developmental biology, gastroenterology, cancer, and stem cells.

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image: Parallel Plagues

Parallel Plagues

By | April 1, 2016

Like cancer, ecological scourges result from the breakdown of regulatory processes, and may be treated with similar logic.

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image: Tumor Traps

Tumor Traps

By | April 1, 2016

After surgery to remove a tumor, neutrophils recruited to the site spit out sticky webs of DNA that aid cancer recurrence.

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A study suggests bats in Asia could have genes that protect them from the fungal infection that is decimating bat populations in North America.

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image: More Support for Allergen-Exposure Strategy

More Support for Allergen-Exposure Strategy

By | March 8, 2016

A second study finds evidence that feeding children peanuts could help prevent them from developing allergies to the legume later in life.

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image: Viral Remnants Help Regulate Human Immunity

Viral Remnants Help Regulate Human Immunity

By | March 3, 2016

Endogenous retroviruses in the human genome can regulate genes involved in innate immune responses.

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image: Amoebae Have Human-Like Immunity

Amoebae Have Human-Like Immunity

By | March 2, 2016

Dictyostelium discoideum produce extracellular nets that can kill bacteria, just as phagocytes in people and other higher animals do, according to a study.

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image: Giant Virus Has CRISPR-like Immune Defense

Giant Virus Has CRISPR-like Immune Defense

By | March 2, 2016

The genome of a mimivirus strain resistant to a virophage has repeated phage sequences alongside nuclease- and helicase-coding sections.

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image: Mutations Not Tied to Metastasis

Mutations Not Tied to Metastasis

By | February 25, 2016

Clinical cases link immune changes to a cancer’s spread through the body, but find no role for so-called “driver” mutations.

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