A new technique could soon spur unprecedented insight into the role of bacterial epigenetics in the evolution of pathogen virulence.
The Scientist
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Weakened viruses used in vaccines can swap genes and produce disease-causing strains.
Field studies reveal non-virulent bacteria take advantage of their virulent counterparts to get a free pass into their host.
To protect themselves during malaria infections, mice can kill their own healthy red blood cells, cutting off the parasite’s primary resource.
As much as rainforests or deep-sea vents, the human gut holds rich stores of microbial chemicals that should be mined for their pharmacological potential.
Microbes, both good and bad, can exert direct effects on host cells and vice versa. For example, pathogenic bacteria such as some strains of E. coli and Salmonella reduce the overall number of normal gut commensal bacteria, promoting their own growth
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