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HHMI pays for open access

The Howard Hughes Medical Institute has decided to pay article processing charges if HHMI-funded scientists choose to publish in open access journals from BioMed Central (BMC). Articles published in BMC journals will be immediately free on the Web. The current BMC linkurl:article-processing charge;http://www.biomedcentral.com/info/about/apcfaq#howmuch ranges from $500 to $2400, depending on the journal. These charges will not come out of an HHMI investigator's budget, so will therefore be in a

By | August 21, 2007

The Howard Hughes Medical Institute has decided to pay article processing charges if HHMI-funded scientists choose to publish in open access journals from BioMed Central (BMC). Articles published in BMC journals will be immediately free on the Web. The current BMC linkurl:article-processing charge;http://www.biomedcentral.com/info/about/apcfaq#howmuch ranges from $500 to $2400, depending on the journal. These charges will not come out of an HHMI investigator's budget, so will therefore be in addition to the amount awarded. HHMI linkurl:recently declared;http://www.the-scientist.com/news/display/53366/ that all funded researchers must make their paper freely available in PubMed by six months after publication. This month, BMC linkurl:announced;http://www.the-scientist.com/news/display/53450/ that Yale University has discontinued their membership, due to increases in fees which Kenny Marone, director of Yale's medical library, says have grown in excess of $30,000 over the past year. In FY06, HHMI spent $538 million on scientific research. It is the second largest biomedical funder in the U.S. HHMI-funded researchers have already published 200 articles in BMC journals, paid for by the investigators. Editor's note (posted August 21): This blog has been updated from a previous version.
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Avatar of: Sergio Stagnaro

Sergio Stagnaro

Posts: 1

August 23, 2007

In my experience, among all other review Editors, BMC Editors accept for publication only articles and Rapid Responses politically correct, but not revolutionary... I fear, for instance, that the article, whose abstract sounds as follows, will never be published...a part from my poor english, of course !!!!\nHere it's:\nTITLE.\nOncogenesis is possible exclusively in individuals Oncological Terrain-positive.\n\nUnfortunately, because the congenital functional mitochondrial cytopathology, which is the "conditio sine qua non" of the most frequent and dangerous human disorders, including malignancy, is overlooked, all current researches, like all that investigate the relation between environmental risk factors and carcinomas, are fundamentally biased. I mean that authors do not consider the existence or assess the seriousness as well as the location of Congenital Acidosic Enzyme-Metabolic Histangiopathy, Oncological Terrain is based on, since they overlook such as mitochondrial cytopathology (1-8). In fact, both environmental risk factors and every drug, as oestrogens, suggested as cancer risk factors, "could" influence some human biological functions and/or bring about different disorders, such as cancers, exclusively in relation to both the presence and intensity of CAEMH-dependent Oncological Terrain and Oncological INHERITED Real Risk in a well-defined site of a biological system. I suggested this overlooked functional mitochondrial cytopathology,described in previous papers, as the genetic factor of common human disorders, and particularly of malignancy (1-8). ("Oncological Terrain",in: www.semeioticabiofisica.it), as far as the onset of a lot of disorders is concerned, including solid as well as liquid malignancies. At this point, I would analogously emphasise the well-known pathological powerful influence of smoking on tissue oxygen supply to all biological systems (3, 4). This effect varies notoriously in prevalence and intensity among individuals in relation to the above-mentioned congenital mitochondrial cytopathology, (2). This both "silent" and dangerous action is easy to evaluate at the bed-side with the aid of a stethoscope. I suggest first investigating (i.e., before whatever research) the presence and intensity of CAEMH in the "tested" population, and soon thereafter assessing prevalence and intensity of the "Oncological Terrain" as well as INHERITED oncological Real Risk, which always develop on the basis of the above-mentioned congenital cytopathology, through newborn-pathological type I, subtype a, "oncological", Endoarterial Blocking Devices, causing microvascular remodelling (1-8). In fact, without this alteration of psycho-neuro- endocrine-immunological system, oncological real risk is based on, oncogenesis is not possible. The importance of the above-mentioned inherited real risk factor should not be overlooked, particularly when we assess a "possible" risk factor for disorders biophysical-semeiotic constitution-dependent (9, 10).\n\n \n1) Stagnaro S., Stagnaro-Neri M.Istangiopatia Congenita Acidosica Enzimo Metabolica. Gazz. Med. It.- Arch. Sci. Med. 144, 423, 1985. \n2) Stagnaro S., Stagnaro-Neri M. Una patologia mitocondriale ignorata: la Istangiopatia Congenita Acidosica Enzimo-Metabolica. Gazz. Med. It. - Arch. Sci. Med. 149, 67 1990. \n3)Stagnaro-Neri M., Stagnaro S. Deterministic Chaos, Preconditioning and Myocardial Oxygenation evaluated clinically with the aid of Biophysical Semeiotics in the Diagnosis of ischaemic Heart Disease even silent. Acta Med. Medit. 13, 109 1997.\n 4) Stagnaro-Neri M., Stagnaro S., Cancro della mammella: prevenzione primaria e diagnosi precoce con la percussione ascoltata. Gazz. Med. It. � Arch. Sc. Med. 152, 447 1993 \n5) Stagnaro-Neri M., Stagnaro S. Introduzione alla Semeiotica Biofisica. Il Terreno Oncologico. Ed. Travel Factory, Roma, 2004. http://www.travelfactory.it/semeiotica_biofisica.htm\n6) Stagnaro S., Stagnaro-Neri M., Oncological Terrain, conditio sine qua non of Oncogenesis, 2004: http://www.gutjnl.com/cgi/eletters?lookup=by_date&days=60 \n7) Stagnaro Sergio. "Genes, Oncological Terrain, and Breast Cancer" World Journal of Surgical Oncology., 2005, http://www.wjso.com/content/3/1/45/comments#205475 \n8) Stagnaro S. Reale Rischio Semeiotico-Biofisico. Ruolo diagnostico e patogenetico dei Dispositivi Endoarteriolari di Blocco neoformati patologici tipo I, sottotipo a) oncologici e b). Ed Travel Factory, Roma, www.travelfactory.it, in press\n9) Stagnaro S. Newborn-pathological Endoarteriolar Blocking Devices in Diabetic and Dislipidaemic Constitution and Diabetes Primary Prevention. The Lancet. March 06 2007. http://www.thelancet.com/journals/lancet/article/PIIS0140673607603316/comments?totalcomments=1\n10) Sergio Stagnaro. Mitochondrial Genome of the Mastodon highlights Human Constitutions. PLOS Biology, (01 August 2007) \nhttp://biology.plosjournals.org/perlserv/?request=read-response&doi=10.1371/journal.pbio.0050207#r1725\n

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