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Assessing Amyloid β
Research suggests a physiological role for the presumed villain
The Scientist 2004, 18(6):25
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Recent work has revealed a potential physiological role for amyloid β, often considered a major culprit in Alzheimer disease (AD) pathology. This suggests that Aβ, an ordinarily upstanding protein, turns bad as the result of a mob-type effect, when the physical buildup into plaques promotes neuronal damage and loss. "There's been a growing awareness over the last three or four years that it's not just a toxic peptide," says Hugh Pearson from the University of Leeds, UK.
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