Research traces the evolution of a gene variant that reduces the risk of Alzheimer’s disease, finding that it originally evolved in response to infectious bacteria.
Early reports that Omicron causes less-severe disease than Delta seem to be borne out, but it’s not yet clear to what extent that’s due to the variant itself versus the populations it’s infecting.
James R. Heath, PhD, President of the Institute for Systems Biology will discuss key single-cell learnings and applications from the large Seattle-based academic pharmaceutical consortium committed to defining molecular mechanisms of SARS-CoV-2 infection and identifying potential prognostic immune biomarkers. Stacey Willard, PhD, from IsoPlexis will discuss how functional phenotyping individual immune cells using IsoPlexis technology edges researchers closer to predicting the risk for severe disease.