A new study confirms reports of membrane-bound RNA molecules and points to their role in neutrophil migration.

Scientists have genetically modified isolated microbes for decades. Now, using CRISPR, they intend to target entire microbiomes.

Editor's Note: This is the second of two articles on the difficulties of proving that a virus contributes to a disease. The first article, on mouse mammary tumor virus and human breast cancer, appeared in the April 17 issue of The Scientist.1 Donald R. Carrigan and Konstance K. Knox Someone once said that if you want to ruin your reputation, go into MS [multiple sclerosis] research, quips Jacqueline E. Friedman, a senior research associate at Rockefeller University. But Friedman, who deals wit

Meritxell Huch tackled her pipedream of growing three-dimensional pancreatic tissue in a dish.

A method for culturing the infectious stage of the Plasmodium lifecycle could increase malaria vaccine production efficiency by tenfold, study authors say.

The antimicrobial drug neomycin protects mice from some viral infections, complicating the picture of the relationship between antibiotics and susceptibility to viruses.

To understand how memories are formed and maintained, neuroscientists travel far beyond the cell body in search of answers.

A gene essential for information storage in the brain engages an autoregulatory feedback loop to consolidate memory.

Damaged lysosomes are repaired by a lipid-based signaling pathway dubbed PITT that could be targeted to treat neurodegenerative disease, its discoverers say.