muscle

In this adapted excerpt from the book, the author relays the past, present, and future of artificial hearts.

Researchers untangle the multifarious nature of muscle aging. So far, the only reliable treatment is exercise.

Researchers boosted levels of the dystrophin protein to nearly normal levels in a canine model of the disease.

The flow of calcium and potassium ions keeps muscles contracting in the diaphragms of neonatal mice, but if a key protein receptor is missing, fatigue sets in more quickly.

A new study reveals more about the role of specialized Schwann cells at junctions between neurons and muscle cells.

Alterations to a protein involved in breathing may help explain some cases of sudden infant death syndrome, a study finds.

Lab-grown muscle stem cells from mice mimic the formation of muscle fibers in vivo. 

Researchers restored muscle function in animals with muscular dystrophy.

These facial muscle cells were genetically reprogrammed into beating heart cells.

An endogenous retrovirus that supports placenta formation in females also helps male mice build muscle, according to a study.

Feedback from muscle spindles and tendon organs provides information about where our bodies are in space and whether or not they are moving.

The cells of the mammalian immune system do more than just fight off pathogens; they are also important players in stem cell function and are thus crucial for maintaining homeostasis and recovering from injury.

Studies reach conflicting conclusions on GDF11 as a rejuvenating factor.

The repeal of a Molecular Endocrinology paper is the second of three anticipated retractions from a cell biologist.

Three groups of researchers used the gene-editing method to restore a protein deficient in Duchenne muscular dystrophy.

The accumulation of fat within skeletal muscle, as happens with obesity, diminishes muscle performance.

An RNA thought to be noncoding in fact encodes a small protein that regulates calcium uptake in muscle.

Previously shown to boost muscle growth in aged mice, a protein’s role in regeneration just got more complicated.

Scientists reveal the neural underpinnings—and muscles tightly linked with—the involuntary flexing of the pelvic floor, which comprises muscles that help us delay urination.