Image of the Day: Fountain of Youth

Neural plasticity wanes with age, but increasing the protein Arc—abundant earlier in life—in the visual cortex of mice can fend off this decline.

By | August 15, 2017

An artificially colored immunofluorescence image depicting labeled Arc protein (green) within the visual cortex of a transgenic mouse engineered to overexpress the Arc gene following exposure to light.ELISSA PASTUZYNArtificially increasing Arc through transgenic overexpression in engineered mice or via viral vectors later in life can prolong and re-establish, respectively, brain plasticity in the visual cortex. 

See K.R. Jenks et al., “Arc restores juvenile plasticity in adult mouse visual cortex,” PNASdoi:10.1073/pnas.1700866114, 2017.

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Avatar of: James V. Kohl

James V. Kohl

Posts: 476

August 16, 2017

Energy is required because proteins do not create themselves and virus-driven energy theft causes the degradation of messenger RNA in all living genera.

The restoration of neural plasticity requires the food energy-dependent pheromone-controlled de novo creation of genes. For example, see: Natural Selection on the Olfactory Receptor Gene Family in Humans and Chimpanzees

Desperate attempts to invent new theories that link trophic coherence to evolution should not be be confused with the facts that have been detailed in works published by serious scientists. See the pseudoscientiific nonsense in this published work: Looplessness in networks is linked to trophic coherence

For comparison: Feedback loops link odor and pheromone signaling with reproduction

There is no desperation manifested in the works of serious scientists who understand the facts about natural selection for energy-dependent codon optimality and de novo gene creation. The energy-dependent creation of new genes restores juvenile plasticity via the creation of G protein-coupled receptors.

 

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