Human prenatal development can be viewed as a program of genetic switches that turn on, in a highly regulated manner, at specific places and times. But a body of evidence is emerging that paints a less hardwired picture, one of responses to environmental challenges fostering changes early on that reverberate decades later, in the guise of cardiovascular disease and diabetes. A symposium at the American Society of Human Genetics annual meeting, held in Philadelphia Oct. 3-7, addressed the fetal origin of adult diseases.
Interested in reading more?
Become a Member of
Receive full access to digital editions of The Scientist, as well as TS Digest, feature stories, more than 35 years of archives, and much more!
Already a member?