Researchers link the ability of the cells to bind and present DNA from pathogens and cell death to anemia, which is common in COVID-19, and immune activation.

If the immune system makes mistakes—reacting late or getting the target wrong—it can amplify the damage wrought by SARS-CoV-2.

Different variants of the virus behind the COVID-19 pandemic are swapping chunks of genetic material, but it’s not yet clear what implications that may have for public health.

Symptoms documented in cases of long COVID are wide ranging and variable.

Every medical intervention—even one with a centuries-long history—brings dangers, some of which become clear only later.

A year and a half into the pandemic, the long-term effects of SARS-CoV-2 infection are garnering more research attention as millions of long COVID patients emerge.

RNA-DNA complexes that were modified with cholesterol made it into the brains of rats and mice, where they knocked down target genes.

A long noncoding RNA from humans appeared to help the enzyme phenylalanine hydroxylase work better in a mouse model of phenylketonuria, the disorder characterized by reduced activity of that enzyme.

For too long, a physiological inability to conceive or carry a child through to birth has been seen as a minor medical issue.

Hearing others’ perspectives on infertility and pregnancy has me reconsidering my own reproductive journey.

Meet some of the people featured in the August 2021 issue of The Scientist.

The three-month moratorium comes after a former prion researcher was diagnosed with Creutzfeldt-Jakob disease.

The AlphaFold program from AI firm DeepMind has amassed a huge database of protein structures from humans and model organisms.