Herpes Viruses Implicated in Alzheimer’s Disease
Anna Azvolinsky | Jun 21, 2018
A new study shows that the brains of Alzheimer’s disease patients have a greater viral load, while another study in mice shows infection leads to amyloid-β build up.
Alzheimer’s-Related Protein Can Spread by Tainted Growth Hormone
Alzheimer’s-Related Protein Can Spread by Tainted Growth Hormone
A hormone treatment contaminated with amyloid-β given to mice caused the protein’s accumulation in their brains, suggesting the same could have occurred in humans given the therapy.
A hormone treatment contaminated with amyloid-β given to mice caused the protein’s accumulation in their brains, suggesting the same could have occurred in humans given the therapy.
Alzheimer’s Should be Characterized by Biomarkers: Report
Diana Kwon | Apr 10, 2018
A proposed definition of the disease emphasizes signs of neurodegeneration and the presence of β-amyloid and tau, rather than cognitive symptoms.
Researchers Develop Potential Blood Test for Alzheimer’s Disease
Catherine Offord | Feb 2, 2018
The test uses levels of plasma amyloid-β to estimate the buildup of protein plaques in the brain.
Do Microbes Trigger Alzheimer’s Disease?
Jill U. Adams | Sep 1, 2017
The once fringe idea is gaining traction among the scientific community.
Infographic: Brain Infection and Alzheimer’s Disease Pathology
Jill U. Adams | Aug 31, 2017
Emerging evidence links bacterial or viral infection with the neuropathology of Alzheimer’s disease.
More Evidence of Alzheimer’s Transmission
Jef Akst | Jan 27, 2016
Examining the brains of seven patients who died of the prion disease called Creutzfeldt–Jakob, researchers find signs of Alzheimer’s pathology.
Delayed Turnover
Amanda B. Keener | Jul 21, 2015
Aggregate-forming amyloid β proteins are replenished more slowly with age, and this may contribute to a person's risk of developing Alzheimer’s disease.
Tangle Trigger
Jyoti Madhusoodanan | Jan 1, 2015
An enzyme that cleaves tau protein in acidic cellular conditions may trigger early events in Alzheimer’s disease.
Shoring Up Golgi To Slow Alzheimer’s
Kerry Grens | Dec 8, 2014
Blocking activity of a kinase in a mouse model protects Golgi in cells and reduces the build-up of amyloid β, a primary component of Alzheimer’s disease.