beta-amyloid
Herpes Viruses Implicated in Alzheimer’s Disease
Herpes Viruses Implicated in Alzheimer’s Disease
Anna Azvolinsky | Jun 21, 2018

A new study shows that the brains of Alzheimer’s disease patients have a greater viral load, while another study in mice shows infection leads to amyloid-β build up.

Alzheimer’s Should be Characterized by Biomarkers: Report
Alzheimer’s Should be Characterized by Biomarkers: Report
Diana Kwon | Apr 10, 2018
A proposed definition of the disease emphasizes signs of neurodegeneration and the presence of β-amyloid and tau, rather than cognitive symptoms.
Researchers Develop Potential Blood Test for Alzheimer’s Disease
Researchers Develop Potential Blood Test for Alzheimer’s Disease
Catherine Offord | Feb 2, 2018
The test uses levels of plasma amyloid-β to estimate the buildup of protein plaques in the brain.
Do Microbes Trigger Alzheimer’s Disease?
Do Microbes Trigger Alzheimer’s Disease?
Jill U. Adams | Sep 1, 2017
The once fringe idea is gaining traction among the scientific community.
Infographic: Brain Infection and Alzheimer’s Disease Pathology
Infographic: Brain Infection and Alzheimer’s Disease Pathology
Jill U. Adams | Aug 31, 2017
Emerging evidence links bacterial or viral infection with the neuropathology of Alzheimer’s disease.
More Evidence of Alzheimer’s Transmission
More Evidence of Alzheimer’s Transmission
Jef Akst | Jan 27, 2016
Examining the brains of seven patients who died of the prion disease called Creutzfeldt–Jakob, researchers find signs of Alzheimer’s pathology. 
Delayed Turnover
Delayed Turnover
Amanda B. Keener | Jul 21, 2015
Aggregate-forming amyloid β proteins are replenished more slowly with age, and this may contribute to a person's risk of developing Alzheimer’s disease.
Tangle Trigger
Tangle Trigger
Jyoti Madhusoodanan | Jan 1, 2015
An enzyme that cleaves tau protein in acidic cellular conditions may trigger early events in Alzheimer’s disease.
Shoring Up Golgi To Slow Alzheimer’s
Shoring Up Golgi To Slow Alzheimer’s
Kerry Grens | Dec 8, 2014
Blocking activity of a kinase in a mouse model protects Golgi in cells and reduces the build-up of amyloid β, a primary component of Alzheimer’s disease.
In Old Blood
In Old Blood
Jef Akst | Aug 1, 2014
The body of a supercentenarian expands science’s appreciation for the physiological limits of aging.