Widening the gap between a bacterium's cell wall and its outer membrane impairs its ability to respond to stress-inducing signals.
© STEVE GRAEPEL
When a bacterium encounters a stressor, RcsF inhibits IgaA, lifting its blockage on the activity of downstream components of the Rcs network. But if the periplasmic distance widens, RcsF is unable to reach IgaA, preventing the system from initiating a stress response.
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Diana is a freelance science journalist who covers the life sciences, health, and academic life. She’s a regular contributor to The Scientist and her work has appeared in several other publications, including Scientific American, Knowable, and Quanta. Diana was a former intern at The Scientist and she holds a master’s degree in neuroscience from McGill University. She’s currently based in Berlin, Germany.
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