Mice with overactive LINE-1 retrotransposons in their brains exhibit movement difficulties, suggesting the genetic elements may play a role in ataxia in humans.
Organelles isolated from two types of neurons and a nonneuronal astrocyte in the mouse cerebellum showed varying levels of proteins, hinting at functional differences.
Over the past decade, researchers at RIKEN Center for Developmental Biology in Japan have generated complex tissues, including mouse retinas and Purkinje cells (a type of neuron) that integrated appropriately into the mouse fetal brain.