Despite the treatment being given for only six weeks, the positive effects lasted for a year.

The researchers say that their results are evidence we haven’t yet reached a limit to human lifespan.

A proposed definition of the disease emphasizes signs of neurodegeneration and the presence of β-amyloid and tau, rather than cognitive symptoms.

Findings from a randomized, controlled trial finds that reducing food intake decreases metabolism and reduces oxidative damage to tissues and cells.

The findings suggest that faster synthesis, rather than decreased clearance, causes the protein to build up in neurons.

Two studies examining the effects of parents’ ages on their offsprings’ telomere lengths come to opposite conclusions.  

The findings contradict what has been observed in humans.  

Scientists identified a gene involved in the age-related deterioration of mouse neural stem cells.

From extending lifespan to bolstering the immune system, the drug’s effects are only just beginning to be understood.