CRISPRed heart muscle cells from humans gain the ability to make a protein missing in Duchenne muscular dystrophy.

By zooming in on a developing mouse heart, scientists are studying whether defects in vasculature contribute to a thin muscle wall.

It’s the latest fallout from an investigation by Harvard and Brigham and Women’s into work overseen by Piero Anversa.

A trial to test whether so-called c-kit cardiac stem cells, first reported by Piero Anversa, could help patients with chronic heart failure has been halted in light of suspicion surrounding his work.

Harvard and Brigham and Women’s Hospital disavow the work by former faculty member Piero Anversa.

A mouse study adds to the growing body of work disputing the ability of progenitor cells to regenerate muscle tissue in adult mammals’ hearts.

Having pioneered the study of muscle physiology in mammals, she uncovered how ion channels enable muscle movement.

Among 11 infants treated to date, most survived and their heart function improved.

The extracellular matrix appears to inhibit regeneration; but scientists debate whether heart muscle really comes back.