A year and a half into the pandemic, the long-term effects of SARS-CoV-2 infection are garnering more research attention as millions of long COVID patients emerge.

Meet Stanford University’s Ronald W. Davis, who pivoted his research to study myalgic encephalomyelitis/chronic fatigue syndrome after doctors diagnosed his son with the disorder.

In Chapter 3, “The Adventure,” author Tracie White meets Whitney Dafoe, a young man imprisoned by a chronic disease.

Ronald Davis of Stanford University changed his focus to research on ME/CFS, the disease formerly known as chronic fatigue syndrome, in a bid to help his son and others like him.

A finding of distinct patterns of gene-regulating RNA snippets in the blood of ME/CFS patients in response to a stress test could pave the way for a diagnostic tool for the condition and help untangle its underlying mechanisms.

The COVID-19 pandemic is sparking renewed efforts to study the underlying causes of this complex, debilitating disease, which might be triggered by the novel coronavirus.

A handful of viruses have been associated with long-term, debilitating symptoms in a subset of those who become infected. Early signs hint that SARS-CoV-2 may do the same.

In a small study, researchers devise a test that could distinguish healthy people from those with the syndrome, also known as myalgic encephalomyelitis.

It has been more than seven months since the National Institutes of Health pledged increased funding for myalgic encephalomyelitis/chronic fatigue syndrome research. Here is how some of that money will be put to use.