Single-cell genome analyses reveal the amount of mutations a human brain cell will collect from its fetal beginnings until death.

Meet some of the people featured in the November 2017 issue of The Scientist.

Every human brain is far more unique, adaptable, and vulnerable than ever suspected.

Advances in single-cell technologies have revealed vast differences between cells once thought to be in the same category, calling into question how we define cell type in the first place.

No two neurons are alike. What does that mean for brain function?

Recent advances in single-cell omics and other techniques are revealing variation at genomic, epigenomic, transcriptomic, and posttranscriptomic levels.

Using a new approach to analyze the transcriptomes of thousands of individual cell nuclei in postmortem brains, researchers identify multiple neuronal subtypes.

Somatic mosaicism may be responsible for a larger proportion of genomic variability within humans than previously thought.

Some genetic abnormalities that appear to have sprung up independently in children are in fact present in a portion of their parents’ cells.

A tree in Australia was found to have genetically dissimilar leaves that varied in attractiveness to herbivores.

Small circles of extrachromosomal DNA appear to be widespread in mammals, and may be byproducts of small deletions in the nuclear DNA of somatic cells.