A molecule produced by a strain of Staphylococcus epidermis interferes with DNA synthesis.

When certain melanocyte stem cells are exposed to UV rays, a molecular cascade can trigger melanoma, scientists find in mice.

Early-stage melanoma cells alter proteins in nearby skin cells to create a favorable environment for cancer progression.

People without red hair who possess the variant are also more likely to develop this form of skin cancer, researchers report.

Rockefeller University researcher Elaine Fuchs on being a woman in science and her contributions to the burgeoning field of reverse genetics

Two compounds boost the ability of melanoma cells to invade other tissues in mice, providing additional evidence that antioxidants can be beneficial to malignant cells as well as healthy ones.

A deep-sequencing analysis reveals that non-malignant skin cells harbor many more cancer-driving mutations than previously expected. 

Researchers uncover a previously unknown way UV light can act on melanin, spurring cancer-causing mutations hours after sun exposure.

A genetic mutation tied to risk of developing testicular cancer may be more prevalent in white men because it also confers a reduced risk of developing skin cancer.

The large mammals alter the levels of melanin in their skin depending on sun exposure, helping them to avoid DNA damage.