The Muscle Issue

Take a break from the bench to puzzle and peruse.

After two decades of failure, novel scientific insights and technical progress are spurring meaningful innovation in the field.

Juan Valverde de Amusco’s anatomical drawings corrected some earlier errors while making anatomy more accessible.

Myosin proteins tug on the cell membrane, giving an erythrocyte its distinct shape.

A protein in skeletal muscles helps mice recover from sleep deprivation.

Even if people exercise, hydrogen peroxide and other reactive molecules continue to accumulate in the tissues.

Having pioneered the study of muscle physiology in mammals, she uncovered how ion channels enable muscle movement.

In this adapted excerpt from the book, the author relays the past, present, and future of artificial hearts.

Meet some of the people featured in the September 2018 issue of The Scientist.

Researchers identify a handy tool for tinkering with the versatile vesicles.

The muscle biologist developed a zebrafish model for myofibrillar myopathies and is working with killifish to study muscle regeneration.

Titin was discovered four decades ago, but some physiology textbooks fail to recognize the important role it plays in muscle contraction.

Scientists recreate proprioception for people with artificial arms using a perceptual illusion.

Isotope changes in the bones demonstrate a similar pattern across far-flung islands.

The presence of aromatic greens in with other building materials increased the time birds sat on their eggs, improving the health of the hatchlings.

Scientists who designed a marine robot to survey ocean life were inspired by eel larvae.

Scientists fiddle with formulas to boost the growth of their stem cell cultures.

Selected Images of the Day from the-scientist.com

Science journalists are essential to advancing the quality of the research enterprise.

What happens when myosin inside red blood cells can't do its job?

Researchers harness a protein to send cargo-carrying exosomes to mouse muscles.

This third filament involved in muscle function is often ignored in medical textbooks. Here's how it works.

Biological insights from the last decade of research on amyotrophic lateral sclerosis are revealing novel opportunities for treatment.

Numerous cellular changes underlie the decline of muscle mass and strength in the elderly.

Timekeepers in muscle help mediate metabolism, and may influence neurological processes such as sleep.

The disease is caused by mutations in a single gene. Can gene editing fix the problem?