Researchers envision a cochlear implant–like device for the nose to give people with impaired olfaction a sense of smell.

In mice, seeing and smelling food causes immediate cellular changes that ready the liver to convert incoming amino acids to proteins.

The compound engages with a receptor in hair follicle cells and prevents skin cells from dying.

The parasite changes people’s scent, primarily due to an increase in aldehydes.

Responses to compounds in human sweat may help explain why people with autism spectrum disorder tend to struggle with social cues.

Scientists demonstrate that just the right amount of inflammation after an injury to a mouse’s olfactory epithelium is key for regenerating cells important for smell.

A study traces proteins’ evolution from the immune to the olfactory system.

Sensory neurons in the tip of the zebrafish nose respond to molecular signals released from food sources.

Animals lacking a sense of smell stayed thinner than their smelling counterparts, despite eating the same amount.