John Connolly and Masataka Nishiga discuss the effects of SARS-CoV-2 on the neurological and cardiovascular systems.

The cholesterol-lowering drugs quell inflammation and reverse endothelial tissue damage, hints that they might curb the body’s excessive immune response to SARS-CoV-2 infection.

Early reports from hospitals document a spike in large vessel blockages, especially among people in their 30s and 40s who tested positive for the coronavirus.

Emerging data as well as knowledge from the SARS and MERS coronavirus outbreaks yield some clues as to why SARS-CoV-2 affects some people worse than others.

A bioengineered heart made of pig and synthetic tissues beats like the real thing.

A study in mice reveals that stem cell transplants, currently in clinical trials, may not actually require the cells.

Scientists show the approach can kill cells that cause hardening of heart tissue in mice.

Researchers seek to untangle the biological mechanisms linking resident microbes to our hearts—and to harness them therapeutically.

A comparison of thousands of former athletes in the two sports finds that NFL players were more likely than MLB players to die from cardiovascular and neurodegenerative diseases, in particular.