cardiovascular disease

He was among the first to identify lifestyle factors that contribute to cardiovascular disease.

The University of Washington researcher leveraged data from the Human Genome Project to identify genes underlying various health conditions and advance precision medicine.

Experts have a decent grasp on how COVID-19 impacts cardiovascular health in the near term. The implications of long COVID, however, remain mysterious.

The procedure, performed last Friday, is a milestone in a long effort to boost the number of organs available for safe transplantation.

The Scientist spoke with Rachel Zeig-Owens, the director of epidemiology for the World Trade Center Health Program, about what scientists have learned after two decades of studying illness and disease among survivors.

Research in mice and humans points to oxidative stress and inflammation as likely drivers of noise-induced health effects such as hypertension and heart disease.

Meet some of the people featured in the June 2021 issue of The Scientist.

Sound from cars, aircraft, trains, and other man-made machines is more than just annoying. It increases the risk of cardiovascular disease.

The FDA and CDC are investigating a handful of reports of blood clots that occurred several days after people received the one-and-done shot.

Mice that experienced heart attacks underwent a large-scale shift in their immune systems that allowed cancer to flourish, perhaps explaining the observation in patients.

The cholesterol-lowering drugs quell inflammation and reverse endothelial tissue damage, hints that they might curb the body’s excessive immune response to SARS-CoV-2 infection.

Early reports from hospitals document a spike in large vessel blockages, especially among people in their 30s and 40s who tested positive for the coronavirus.

Emerging data as well as knowledge from the SARS and MERS coronavirus outbreaks yield some clues as to why SARS-CoV-2 affects some people worse than others.

A bioengineered heart made of pig and synthetic tissues beats like the real thing.

A study in mice reveals that stem cell transplants, currently in clinical trials, may not actually require the cells.

Scientists show the approach can kill cells that cause hardening of heart tissue in mice.

Researchers seek to untangle the biological mechanisms linking resident microbes to our hearts—and to harness them therapeutically.